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Dental Abnormalities in Children Affected with Hyperbilirubinaemia | ||
| Tikrit Journal for Dental Sciences | ||
| Volume 4, Issue 1, June 2016, Pages 22-28 PDF (204.49 K) | ||
| DOI: 10.25130/tjds.4.1.5 | ||
| Author | ||
| Salim Jasim Khalaf | ||
| (1) Lec., Tikrit University, College of dentistry, Basic Scinece/ Microbiology Department. | ||
| Abstract | ||
| This study was carried out to evaluate the relationship between hyperbilirubinaemia and dental abnormalities (D.A). Group of patients composed of 52 patient, and control group composed of 35 healthy child. This study was conducted on children that ages between (6-12 yeas). The markers measured in this study were total serum bilirubin (TSB) (conjugatedand unconjugated), alanine amino-transferase (ALT), gamma-glutamyltransferase (GGT),calcium (Ca), and vitamin D.This study show a significant decrease (P<0.05) in levels of serum calcium and viamin D, a significant increase (P<0.05) in levels of serum ALT and GGT in patients as compared with healthy persons,and also these changes proportional with hyperbilirubinaemia due to conjugatedtype increase (normal unconjugated type), also dental abnormalitiesproportional with these changes.Bilirubin is a byproduct of heme catabolism from aged redblood cells.Bilirubin is primarilyproduced in the liver,spleen, and bonemarrow. Total bilirubin is the sum ofunconjugated bilirubin,monoglucuronide and conjugated bilirubin, bounddelta bilirubin(1). Alanine transaminase diglucuronide, and albumin (ALT) is acytosolic enzyme which is specific for liver function examination.ALT is found in large amounts in the liver, and small amounts of this enzyme are also found in the heart, muscle, and kidney. Gamma glutamyltransferase (GGT) is a cell surfaceprotein contributing extracellular (GSH(.catabolismof to the glutathione (1) Lec., Tikrit University, College of dentistry,Basic Scinece/ Microbiology Department.The enzyme is produced inmany tissues,but most GGT in serum is derived from theliver(2,3). | ||
| Keywords | ||
| Dental abnormalitie Hyperbilirubinae mia | ||
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